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organizing pneumonia

Synopsis

There is transformation of intraalveolar exudate into intraalveolar fibro-myxoid nodular masses containing firoblast and chronic inflammatory cells. Corresponds to grey hepatization of lobar pneumonia.

- intraluminal organizing fibrosis in distal airspaces

- patchy distribution (pulmonary nodules)
- preservation of lung architecture
- uniform temporal appearance
- mild interstitial chronic inflammation
- negative elements

  • lack of interstitial fibrosis (except incidental scars or apical fibrosis)
  • lack of granulomas
  • lack of neutrophilic infiltration or abcesses
  • lack of necrosis
  • lack of hyaline membranes or prominent airspace fibrin
  • lack of prominent eosinophilic infiltration

Variants

- diffuse organizing pneumonia
- localized organizing pneumonia

Etiology

- organizing pulmonary infections

  • Pneumocystis carinii pneumonia

- organizing diffuse alveolar damage
- drug reaction (drug-associated organizing pneumonia)
- toxic-associated organizing pneumonia

  • cocaine abuse

- dysimmune diseases/auto-immune diseases (collagen vascular diseases, connectivitis)
- HIV infection (HIV-associated organizing pneumonia)
- viral infections (virus-associated organizing pneumonia)

- hypersensitivity pneumonitis (extrinsic allergic alveolitis)
- chronic eosinophilic pneumonia
- myelodysplastic syndrome
- ionizing radiations
- pulmonary hemorrhages
- pulmonary vasculitis

- airway diseases complicated by infections

- pulmonary airways obstruction
- peripheral reaction around

- bronchiolitis obliterans organizing pneumonia
- acute fibrinous organizing pneumonia
- cryptogenic organizing pneumonia (COP) (or cryptogenic BOOP)

Differential diagnosis

- UIP pattern

- acute interstitial fibrosis (AIP)
- DAD pattern

- NSIP pattern (cellualr pattern of NSIP)
- acute fibrinous and organizing pneumonia (AFOP)
- eosinophilic pneumonia (if eosinophilic infiltration)